Cerebral angiography was performed on eight patients. Six patients were evaluated by an extensive neuropsychological battery. The most frequent symptoms of caudate hemorrhage were headache and/or vomiting, and decreased consciousness. Clinical features were similar to those of subarachnoid hemorrhage.

Basal Ganglia (Caudate) Intracerebral Hemorrhage Note the large intracerebral hemorrhage originating in the area of the caudate nucleus on the right. Also, note that the hemorrhage is forming a “cast” of much of the anterior lateral ventricle on the right. The hemorrhage has spread to the adjacent lateral ventricle.

Introduction. Bleeding within the fixed vault of the cranium is a life-threatening emergency that affects around 65,000 patients a year in the United States of America. Intracranial hemorrhage can cause increased intracranial pressure, which in turn damages the brain parenchyma and can cause herniation.

Like the lateral ventricles, the caudate is a C-shaped structure with a thick anterior portion called the head, which becomes narrower as it extends towards the back of the brain. The middle portion of the caudate is known as the body, and this tapers off into the tail of the caudate.

The head of the caudate nucleus is supplied by the recurrent artery of Heubner, a small branch for the A1 (or sometimes the A2) segment of the anterior cerebral artery.

The clinical presentation of CN hemorrhage is often characterized by a clinical presentation mimicking subarachnoid hemorrhage, while clinical features of both ischemic and hemorrhagic strokes included behavioral abnormalities dysarthria, movement disorders, language disturbances and memory loss.

The nucleus caudatus is not a common location of spontaneous intracerebral hemorrhage. Twelve patients (8 men and 4 women) aged 38-76 years who had caudate hemorrhage between November 1, 1992 and March 31, 1994 were evaluated.

Fifteen patients with caudate infarct (60%) and 3 patients with hemorrhage (50%) were able to return to normal daily life.